A good question and well done for admitting that you don’t know about it or understand what hip dysplasia in dogs is.
The hip joints are at the top of the hind leg in dogs and are ‘ball and socket’ joints, so that flexion (lifting the knee up and forward), extension (moving the straight leg back the same way) and rotation (think squatting to pee) are all possible.
The joint is formed by the round ‘head’ of the femur (or thigh bone) inserting into the acetabulum of the pelvis, which is the ‘socket’. Some stability is added by the presence of the ‘round’ ligament, which attaches the femoral head to the acetabulum, and the joint capsule that surrounds the joint.
What we would want in an individual is a nice, round ball fitting snugly into a nice, round socket. But, of course, sometimes things can go wrong.
Despite what some breeders will tell you, hip dysplasia is considered an inherited trait. However, it is also influenced by environmental factors.
Severe hip dysplasia with significant remodelling of the joint. This dog was sound with only restricted movement evident.
If in a young, growing animal, the soft tissue surrounding the joint fails to support it properly, then laxity of the joint will occur. Depending on the degree of movement, a destructive chain of events follows:
• Inflammation in the joint capsule causes pain
• Joint fluid increases, further destabilising the joint
• The bones move together abnormally causing erosive damage
• The round ligament is stretched allowing even more abnormal movement
• Lameness results in poor muscle development
• Tiny fragments of bone float in the joint (joint mice)
• Remodelling of the femoral head and neck occur
• The femoral head flattens and the neck thickens
• In less severe cases, stability returns but movement is restricted
• Further deterioration after maturity
Clinical signs of hip dysplasia in dogs are usually obvious but stalwart dogs can appear normal until their gait is examined closely. Affected dogs may be reluctant or slow to rise, climb stairs and/or jump. Intolerance of exercise is common. There is pain on extension of the hip, especially when it is also rotated outwards. Well-muscled forequarters can cast some suspicion, as weight is thrown forward to protect the hip. When both hips are affected, you may see a ‘bunny hop’ gait and a ‘kyphotic’ posture (forward rounding of the lower back). Diagnosis can be confirmed by X-ray, which must be done under general anaesthesia or heavy sedation, so that luxation (dislocation) of the hips can be assessed and specific tests (Barden and Ortolani signs) be undertaken. It must be noted, however, that there is no correlation between the severity of the radiological changes and the degree of lameness. Some working dogs seem pretty resistant to the effects of hip dysplasia but care should be taken that these individuals are not bred from. And no matter what anyone says, it is just not possible to assess the quality of the hip joint without X-rays.
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Sometimes you need a bit of luck, but it helps to source pups from tested parents that have good ‘hip scores’. The BVA Hip Dysplasia Scheme calculates a score for each hip. Most dogs require anaesthesia to allow a perfectly straight ventro-dorsal X-ray of the hip joints to be taken. The best score per side is 0 and the worst 53. Thus perfect hips score 0-0. Only dogs with better than Breed Mean Score hips should be bred from. In Labradors, this would be significantly better than a total score of 18. The results for individual dogs can be viewed on the Kennel Club’s website.
Many affected young dogs will, with conservative management, weight loss and anti-inflammatory treatment, return to being sound but they will rarely be able to work successfully. In older dogs, progressive arthritis generally occurs, requiring medical input.
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